Diamyltryptamine
| File:Diamyltryptamine.svg | |
| File:Diamyltryptamine 3D.png | |
| Clinical data | |
|---|---|
| Other names | DAT; DAmT; N,N-Diamyltryptamine; Dipentyltryptamine; N,N-Dipentyltryptamine; DPeT |
| ATC code |
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| Pharmacokinetic data | |
| Onset of action | Unknown[1] |
| Duration of action | Unknown[1] |
| Identifiers | |
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| E number | {{#property:P628}} |
| CompTox Dashboard (EPA) |
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| ECHA InfoCard | {{#property:P2566}}Lua error in Module:EditAtWikidata at line 29: attempt to index field 'wikibase' (a nil value). |
| Chemical and physical data | |
| Formula | C20H32N2 |
| Molar mass | 300.490 g·mol−1 |
| 3D model (JSmol) | |
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Diamyltryptamine (DAT), also known as N,N-dipentyltryptamine, is a chemical compound of the tryptamine family related to dimethyltryptamine (DMT).[1][2] It is part of the homologous series of tryptamines that includes DMT, diethyltryptamine (DET), dipropyltryptamine (DPT), dibutyltryptamine (DBT), DAT itself, and dihexyltryptamine (DHT).[1][3][4]
Use and effects
[edit | edit source]The compound was briefly mentioned by Alexander Shulgin in his 1997 book TiHKAL (Tryptamines I Have Known and Loved), but he does not appear to have synthesized or tested it.[1] Relatedly, the properties and effects of DAT are unknown.[1] However, it is known that whereas DMT, DET, and DPT are fully effective psychedelics, DBT showed only weak psychedelic effects and DHT was inactive.[3][4][1]
Chemistry
[edit | edit source]Analogues
[edit | edit source]N-Amyltryptamine
[edit | edit source]
The N-monoamyl analogue of DAT, N-amyltryptamine (NAT), has also been described.[1][5] According to Stephen Szara and Alexander Shulgin, this compound was inactive at a dose of up to 100 mg orally.[1][5]
History
[edit | edit source]DAT was first described in the scientific literature by Stephen Szara and colleagues in 1962, who studied its metabolism (specifically 6-hydroxylation) in vitro.[2]
See also
[edit | edit source]References
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